For decades, Alzheimer’s disease has been framed as a disorder caused by toxic protein buildup in the brain—especially beta-amyloid plaques. But groundbreaking research now suggests we may have been looking at the disease from the wrong angle all along.
After more than 30 years of study, scientists propose that Alzheimer’s isn’t primarily a brain disorder, but an autoimmune disease. Beta-amyloid, long blamed as the villain, is actually a normal and crucial part of the brain’s immune defense. It helps protect the brain from infections and injury.
The problem arises when beta-amyloid becomes confused. Certain bacteria share structural similarities with brain cells, causing beta-amyloid to misidentify the brain’s own tissue as a threat. Over time, this triggers a slow, self-directed immune attack—damaging neurons and leading to the cognitive decline we recognize as Alzheimer’s.
We share very few sentiments with our government.
"Over time, this triggers a slow, self-directed immune attack"
This seems to be a common characteristic of these NWO "diseases" crippling the population: something causing the body to work against itself.
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This immune-based framework reshapes our understanding of dementia. Instead of trying to completely remove beta-amyloid—a strategy that has repeatedly failed in clinical trials—future treatments may focus on regulating the immune response, much like therapies for autoimmune diseases such as lupus or multiple sclerosis.
If confirmed, this shift could open the door to safer, more effective treatments—and even preventive strategies—by calming the brain’s immune system rather than fighting one of its natural defenses. It’s a powerful reminder that sometimes, disease isn’t caused by a broken system, but by one that has lost its balance.
Source:
Donald Weaver, Professor of Chemistry and Director of the Krembil Research Institute, University Health Network, University of Toronto. Published in The Conversation.
