Hi Mac or a Fren
Urgently looking for post on ivermectin dosage, its going to close friend for cancer, dr bs not working.

Prayers ..

All possible dosages and drug forms may not be included here. Your dosage, drug form, and how often you take the drug will depend on:

your age
the condition being treated
how severe your condition is
other medical conditions you have
how you react to the first dose
Drug forms and strengths

Generic: Ivermectin

Form: oral tablet
Strength: 3 mg
Brand: Stromectol

Form: oral tablet
Strength: 3 mg
Dosage for parasite infection in the intestinal tract

Adult dosage (ages 18–64 years)

Typical dosage: 200 mcg/kg of body weight taken as one dose. Most people won’t need more than one dose.
Child dosage (ages 0–17 years)

For children who weigh 33 pounds (15 kg) or more

Typical dosage: 200 mcg/kg of body weight taken as one dose. Most children won’t need more than one dose.
For children who weigh less than 33 pounds (15 kg)

It has not been confirmed that this drug is safe and effective for use in these children.

Senior dosage (ages 65 years and older)

Your liver may not work as well as it used to. This can cause your body to process drugs more slowly. As a result, more of this drug can stay in your body for a longer time. This raises your risk of side effects.

Dosage for parasite infection in skin or eyes

Adult dosage (ages 18–64 years)

Typical dosage: 150 mcg/kg of body weight taken as one dose.
Follow-up treatment: You’ll likely need follow-up care from your doctor and additional rounds of treatment with this drug. Your doctor will decide when you’ll receive your next dose of ivermectin. You may be treated again in as few as three months.

Child dosage (ages 0–17 years)

For children who weigh 33 pounds (15 kg) or more

Typical dosage: 150 mcg/kg of body weight taken as one dose. Most children won’t need more than one dose.
Follow-up treatment: Your child will likely need follow-up care from your doctor and additional rounds of treatment with this drug. Your doctor will decide when your child will receive their next dose of ivermectin. Your child may be treated again in as few as three months.
For children weighing less than 33 pounds (15 kg)

It has not been confirmed that this drug is safe and effective for use in these children.

Senior dosage (ages 65 years and older)

Your liver may not work as well as it used to. This can cause your body to process drugs more slowly. As a result, more of this drug can stay in your body for a longer time. This raises your risk of side effects.

As opposed to:

Background: The relationship between vaccines and neuroinflammation have consistent molecular biology bases. In a recent paper we have already analyzed this kind of relationship.

Hypothesis: In this paper, we have gained additional evidence to support the link between vaccines and neuroinflammation. Furthermore, we found the molecular bases that support the link between HPV vaccines and certain adverse events (AEs). The peripheral proinflammatory cytokines (IL-1β, IL-6, and TNF-α), expressed after the injection of the vaccines can reach the brain and can cause neuroinflammation after microglia activation.

After vaccine injection significant systemic immune activation may occur with signs suggesting reactive brain inflammation, such as acute crying, fever, restlessness and failure to eat. It is a warning of danger to the brain in front of which we should reflect before causing irreversible damage. We also hypothesized the existence of a post-vaccination inflammatory syndrome caused by the proinflammatory cytokines strongly expressed after HPV vaccine injections. In addition, the molecular explanation of the chronic pain that has affected many girls in the world, including the complex regional pain syndrome (CRPS) in Japanese girls.

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Conclusion: All vaccines can cause neuroinflammation. HPV vaccines can cause a post-vaccination inflammatory syndrome characterized by chronic pain and neuroinflammation. In this case, the phenomena of central sensitization is responsible for all the symptoms associated with chronic pain. The strong expression of proinflammatory cytokines, secreted after HPV vaccinations, brings to process that can produce irreversible neurological results in HPV vaccinated girls.

Oxygen radicals and nitric oxide (NO) are generated in excess in a diverse array of microbial infections. Emerging concepts in free radical biology are now shedding light on the pathogenesis of various diseases. Free-radical induced pathogenicity in virus infections is of great importance, because evidence suggests that NO and oxygen radicals such as superoxide are key molecules in the pathogenesis of various infectious diseases.

Although oxygen radicals and NO have an antimicrobial effect on bacteria and protozoa, they have opposing effects in virus infections such as influenza virus pneumonia and several other neurotropic virus infections. A high output of NO from inducible NO synthase, occurring in a variety of virus infections, produces highly reactive nitrogen oxide species, such as peroxynitrite, via interaction with oxygen radicals and reactive oxygen intermediates.

The production of these various reactive species confers the diverse biological functions of NO. The reactive nitrogen species cause oxidative tissue injury and mutagenesis through oxidation and nitration of various biomolecules. The unique biological properties of free radicals are further illustrated by recent evidence showing accelerated viral mutation by NO-induced oxidative stress. NO appears to affect a host's immune response, with immunopathological consequences. For example, NO is reported to suppress type 1 helper T cell-dependent immune responses during infections, leading to type 2 helper T cell-biased immunological host responses. NO-induced immunosuppression may thus contribute to the pathogenesis of virus infections and help expansion of quasispecies population of viral pathogens.

Broad-spectrum and effective antiviral drugs are difficult to come by, and many of the most deadly viruses still do not have vaccines to provide protection in humans. In new work led by David Lembo, Francesco Stellacci and colleagues, they have taken advantage of a mechanism in viruses for cell interaction, combined with nanoparticles, to make a new broad-spectrum antiviral.

The team designed nanoparticles with long linkers that mimic heparin sulfate proteoglycans (HSPG), a conserved surface protein used by viruses in the first steps to binding to cells for infection. Testing the nanoparticles in several in vitro and in vivo assays, the team shows that they bind strongly to a wide range of viruses and effectively inhibit them from infecting cells.

The most basic method to measure the size of nanoparticles is the size analysis from the picture image using the transmission electron microscope (TEM), which could also give the particle size distribution. For this analysis, preparation of the well-dispersed particles on the sample mount is the key issue.

The wavelength corresponding to the maximum extinction, absorption as well as scattering redshifts (shift to longer wavelengths) were observed as the nanoparticle size increased. The rate of change of scattering (∆sca) and absorption (∆abs) relative to the extinction is calculated to correlate these two properties.

Gold nanoparticles’ interaction with light is strongly dictated by their environment, size and physical dimensions. Oscillating electric fields of a light ray propagating near a colloidal nanoparticle interact with the free electrons causing a concerted oscillation of electron charge that is in resonance with the frequency of visible light.