Mitochondrial health is an indicator of overall cell health

Healthy mitochondria exhibit a difference in electrical potential across the lipid bilayer that surrounds them, and some fluorescent dyes will accumulate inside mitochondria as long as this potential difference is maintained.

https://www.thermofisher.com/uk/en/home/life-science/cell-analysis/cell-analysis-learning-center/molecular-probes-school-of-fluorescence/imaging-basics/labeling-your-samples/mitochondrial-labeling.html

Alcohol adversely affects mitochondria. In the USA beer has NOT experienced inflation. Why is that?

Sulfur dioxide blocks nerve signals from the pulmonary stretch receptors and abolishes the Hering–Breuer inflation reflex.

Wow. That why peeps can't breathe after the jab????

The chest wall can become infected by bacteria or viruses.

Bacteria, viruses, and fungus may infect the chest wall, resulting in chest wall pain and inflammation.

Pleurisy (PLOOR-ih-see) is a condition in which the pleura — two large, thin layers of tissue that separate your lungs from your chest wall — becomes inflamed. Also called pleuritis, pleurisy causes sharp chest pain (pleuritic pain) that worsens during breathing.

Ya.... Something else happens w the jab. Sudden Adult Death??? Cmon.... What causes the apnea? Also note more lower extremity edema and leg ulcers... I see it w elderly jabbed I work with.

High levels of particulate matter in ambient air are associated with increased respiratory and cardiovascular health problems. It has been hypothesised that it is the ultrafine particle fraction (diameter

Examination of the bronchoalveolar lavage fluid revealed a small increase in polymorphonuclear cell number (ranging 0.6–1%) after UFCP inhalation, compared with clean air controls, suggesting a minor inflammatory response. However, DNA microarray profile analysis revealed a clearly biphasic response to particle exposure. After 4 h of inhalation, mainly heat shock proteins were induced, whereas after 24 h, different immunomodulatory proteins (osteopontin, galectin-3 and lipocalin-2) were upregulated in alveolar macrophages and septal cells.

In conclusion, these data indicate that inhalation of ultrafine carbon particles triggers a biphasic pro-inflammatory process in the lung, involving the activation of macrophages and the upregulation of immunomodulatory proteins.